晨间信号Morning Signal
《科学》 第391卷 第6790期 · 2026年3月12日 · 中文解读

聚合酶捕获机制揭示H5高致病性禽流感病毒起源

Polymerase trapping as the mechanism of H5 highly pathogenic avian influenza virus genesis · M. Funk et al.
约 109 分钟Research Article Summaries在小程序里点播,20 到 60 分钟做好
这篇讲什么
本文揭示了H5高致病性禽流感病毒通过聚合酶捕获机制获得多碱性切割位点的分子机制。
原文开头
Full article and list of author affiliations: https://doi.org/10.1126/ science.adr6632 Mathis Funk, Monique I. Spronken, Roy M. Hutchinson, Benoit Arragain, Pauline Juyoux, Theo M. Bestebroer, Anja C. M. de Bruin, Alexander P. Gultyaev, Ron A. M. Fouchier, Stephen Cusack, Aartjan J. W. te Velthuis, Mathilde Richard* INTRODUCTION: Influenza viruses pose a continuous health threat to animals and humans worldwide. Outbreaks of highly patho- genic avian influenza viruses (HPAIVs) have a devastating impact on domestic and wild bird populations as well as on wild mam- mals. Moreover, HPAIVs present a health risk to humans through repeated spillover events, renewing concerns about the potential for a new pandemic. …
摘自《科学》(Science)第391卷 第6790期 · 2026年3月12日,M. Funk et al.。仅引用开头一小段供了解文章,版权归原刊所有,全文请阅读原刊。
晨间信号小程序码
微信扫码,在小程序里听完整版
不用登录先听一篇 · 或在微信搜索小程序 晨间信号
同期其他文章